By D. Virgil Alfaro III MD, Peter E. Liggett MD, William F. Mieler MD, Hugo Quiroz-Mercado MD, Rama D. Jager MD MBA FACS, Yasuo Tano MD
A popular team of retina surgeons offers promising new advancements in age-related macular degeneration, with emphasis at the most important advances long ago 5 years. content material addresses all elements of administration, together with anatomy, body structure, and pathophysiology; imaging of the macula in age-related macular degeneration; prevention measures and therapy; therapy; and destiny instructions. Abundantly illustrated and fantastically geared up, this booklet serves as an exceptional reference and textbook.
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Additional resources for Age-related macular degeneration : a comprehensive textbook
B. P21 retina exposed to hyperoxia from P7 to P12, before return to room air. Arrows indicate neovascular tufts extending into the vitreous. Arrowheads mark enlarged intraretinal vessel profiles. GCL, ganglion cell layer; ILM, internal limiting membrane; IPL, inner plexiform layer; V, vitreous (original magnification ×100). (Reprinted with permission from Smith LEH, Wesolowski E, McLellan A, et al. Oxygen-induced retinopathy in the mouse. Invest Opthalmol Vis Sci. 19 closed colony of semi-free-ranging rhesus monkeys that have been maintained in isolation since 1938 by the Caribbean Primate Research Center (CPRC) (87,88).
Photochem Photobiol. 1987;46:881-886. 34. Miller H, Miller B. Photodynamic therapy of subretinal neovascularization in the monkey eye. Arch Ophthalmol. 1993;111:5,855-5,860. 16. Van der Zypen E, Fankhauser F, Raess K. Choroidal reaction and vascular repair after chorioretinal photocoagulation with the freerunning neodymium-YAG laser. Arch Ophthalmol. 1985;103:580-589. 35. Moulton RS, Walsh AW, Miller JW, et al. Response of retinal and choroidal vessels to photodynamic therapy using benzoporphyrin derivative monoacid.
The greatest neovascular response occurred during P17 to P21 (Fig. 4). This was then followed by slow regression of the new vessels with reestablishment of a more normal, branching vascular pattern, which was observed in retinal flat mounts by P24. Quantification of the neovascular response was done by counting vascular nuclei in paraffin cross sections of retina. In addition, retinal flat mounts after fluorescein-dextran perfusion and glial fibrillary acidic protein (GFAP) immunohistochemistry were also used to quantify and confirm neovascularization (80).